Previous and our latest laboratory results showed that
this disease is
caused by filamentous biofilm bacteria (FBB) and subsequently
various secondary infections. Most likely, in the case of an FBB
infection, there is a causal relation to stress-related pre-existing
conditions due to an insufficient, endocrine HPA stress axis.
In particular, noticeable by an overreactive immune system
(stress-induced), whereby an excess of histamine, prostaglandins,
leukotrienes, cytokines, or T-cells are detectable.
Which in the later
course of the disease almost always cause hyperallergic and
multi-inflammatory reactions. Often also blood clots, thrombosis, heart
attacks and ultimately cancer.
In the initial phase of infection by filamentous biofilms (FBB), a
medical diagnosis is extremely difficult. Since a bacterial infection
with biofilms is generally a new dimension of disease, it should force
physicians to rethink.
Especially with this disease, various bacteria of
different kind, which are embedded in a biofilm, can also exchange DNA
with each other via plasmids.
Furthermore, filamentous growth (three-dimensional biofilm) can spread
by means of an extracellular, polysaccharide matrix (biofilm streamers)
consisting of polysaccharides (cellulose), lipids, various proteins and
within endospores or bacterial protoplasm is included with the nucleus and nucleic
acid and the DNA. Some information and images about biofilm streamers - see
Source: Some Information and Images
Bacterial solutions to multicellularity: a tale of biofilms, filaments
and fruiting bodies
This rare filamentous form of biofilm, which spreads
rather fungus-like to colonize the environment, has an evolutionary
individual bacterial accumulations, e.g. free-living bacteria or
so-called planktonic cells, which can only spread obligate and
glide in a limited location, e.g. in any moisture.
In addition, they have extremophilic properties and have found a
survival niche at the boundary between wet and dry areas, and are
multiresistant to common antibiotics. Furthermore, such biofilm
bacteria have been for a long time evolved into multicellular organisms. Such
do not come from Mars, but from the soil! Biofilm
infections are under the radar and could probably be a cause of
Biofilm streamers cause catastrophic
disruption of flow with consequences for environmental and medical
These bacteria are not typical pathogens per se, perhaps opportunistic,
so that they would not only infect immunocompromised individuals, but rather highly sensitive individuals are affected with a stress-related,
over-shooting immune system, healthy people can serve as carriers, but they don't get sick, or not so quickly.
In other words, stress-ridden people and animals are affected, which
probably have a higher production of stress hormones and thus produce
etc, which causes these bacteria to settle; even though animals and
humans are not typical hosts for these nitrogen-binding bacteria, which
are more likely to be found in the soil.
In the later course of the disease the HPA-stress axis will collapse
producing even less stress hormones (cortisol, noradrenalin, etc.), unable to counteract properly the stressful situation anymore, as a
result of a pituitary gland and following adrenal gland insufficiency.
These bacteria are now unfortunately also increasingly marketed as
bacteria for agriculture by biotech companies, such as biological
pesticides, plant growth promoters (nitrogen binding), horizontal gene
transfer (glyphosate plants), etc.
These laboratory tests
should mainly be done if the disease has been present for a longer time!
blood profile (CBC), ACE levels, inflammatory parameters (C3, C4, SAA,
hemoglobin levels, red blood cells, serum albumin, ESR 32, vitamin B1,
B6, B12, vitamin D, serotonin, folic acid, iron, copper, cancer
prevention tests (LDH, etc.)
tests, CRH, ACTH, cortisol, norepinephrine, DHEA, somatropin, cystatin
C, D-dimers, histamine, prostaglandins, leukotrienes, cytokines, NKT, T
cells (CD3, CD4, CD8), autoimmune disease (AMA, ANA, pANCA, cANCA),
collagen, common metabolic values, thyroid gland (TSH, FT3,
Anti-DPO, TRAK), MRI pituitary and adrenal gland
examinations, general Liver values/pancreas, gastric, intestinal
mirrors, small intestine, intestinal villi, stool and urine
examination, calprotectin measurement, enteritis, ulcer, etc.
examinations, autoimmune diseases, myocarditis (M7-AK), chronic
inflammatory diseases of the CNS, carpal tunnel syndrome, examination
of the cranial nerves, nerve conductivity tests
possible common infection parameters (immunoglobulins, C-RP,
procalcitonin, CA50, C1q, IL6), even if some will be initially negative
Biofilm mass growing out from a
black filament (wildlife form)
one is infected with such bacteria, it will arise in the further
course of the typical symptoms as with other bacterial infections, but
by means of serology, no signs of infection can be measured by common
infectious parameters (IgA, IgM, IgG, IgE, etc.), as these bacteria are
embedded in a biofilm and would therefore not present antigens.
The granulocyte levels in the blood may be reduced, or normal, or also
strongly reduced in the later course of the disease.
Also, a disturbed or
decreased hormone balance due to long-term stress (somatropin,
serotonin, cortisol, DHEA, norepinephrine) may be present.
Functional tests CRH/ACTH trigger hormones, as well as haemoglobin
levels, serum albumin and ESR 32 should also be investigated. Sometimes
only leukocytes or CRP (C-reactive protein) levels are slightly
increased or the levels of cytokine, histamine in the blood initially
increased slightly at the beginning of the disease and later borderline.
Procalcitonin is inconspicuous, just as applied blood cultures by
default, are also negative. Even with PCR tests, you can't get any
further, because you don't have the naked bacterium in your blood.
biofilm bacteria usually escape the usual laboratory examination, which
is a disaster for the respective patient.
cultures of biofilm and its
filamentous extensions (streamers) or tissue samples should be made on
MacConkey, blood/urea, broth, nutrient-rich boullions, or bacilli agar
at 26 - 28° Celcius (78,8 °F -
for 6-8 days to detect all involved bacteria inside biofilms, e.g. from the Bacillus cereus group or various other agrobacteria such as Rhizobium radiobacter, A. vitis,
B. megaterium, B. subtilis, B. pumilus, B. mycoites, B. licheniformis, B. circulans, or bacteria like Sphingomonas xenophagum,
Pseudomonas or Stenotrophomonas
In the case of a glucose medium that is rather unsuitable,
one should rather incubate it up to 10 days. R. radiobacter and
some other bacteria are oxidase and urease positive. For an urease test
you can use e.g. test kits for H.
pylori detection ($30 dollars). For this purpose, however,
filaments are used with the test kit.
If you want to go deeper, you could also have phenotypical tests done
after cultivation to determine the biovars. To see what genetic
are dealing with in order to determine the use, or genetic
manipulation, and thus the possible manufacturer.
Genotype identification should not necessarily be done by PCR tests,
but rather via 5'-end gene sequencing via a 16S rRNA test (cost $50-80
dollars), to prove e.g. bacilli or C58,
A. rhizogenes K1026 or K84 (old. A. radiobacter K84), A. radiobacter
or A. vitis LMG 8750T
Even an infrared spectrometry, or an immunofluerence test,
detect rare microorganisms even in previous, negative cultures. It is
also possible to search specifically for elevated values of
polysaccharides (biofilm) through special laboratories.
Evidence of microscopic examinations usually shows only cellulose
filaments with slime produced by these bacteria for attachment to any
host organism, and are confused with clothing fibres, since they also
consist of polysaccharides like cellulose (cotton fibers).
This disease is more likely to be considered a systemic disease and
is not a skin disease per se, as the biofilm usually spreads first from
intestine to the bloodstream, organs and much later on the skin. Later,
immune system reacts more and more with hyperallergic and
multi-inflammatory reactions to the bacterial proteins (proteolytic
digestive enzymes) or by-products (ammonium), and at least in the later
course of the disease some increased inflammatory parameters can be
detected, such as prostaglandins, leukotrienes, cytokines, T cells and
In terms of skin symptoms, there are sometimes more and sometimes fewer
pustules, swellings, blisters or redness, which resemble insect bites.
Probably due to the bacterial enzyme release with subsequent allergic
reaction of the skin (histamine release, etc.), which is not an
IgE-induced reaction. Likewise, these bacteria themselves also produce
histamine, which can later lead to histaminosis or histamine
intolerance, and ammonium, which can lead to convulsions and
this infection with biofilms is systemic, a simple skin assessment
should not serve as the only criteria for definitive disease detection.
As the infection/infestation is mainly still present in the
skin pores, or subcutaneously in deeper skin layers, gastrointestinal
tract, lymphsystem and lymph nodes, - only after weeks or months
manifests itself with the typical skin symptoms, which can resemble
craters, anthrax-like wounds.
also many cases where skin symptoms are more likely to be
reduced, presumably due to a generally lower immune response, histamine
production of the sufferer.
Any suspicion of a
biofilm infection, stool and urine should also
be examined. Dermatologically the whole body, hair areas, outer upper
arms, shoulder parts, lumbar area, inner and outer thighs and also the
eye pockets and nasal and ear sockets by cotton swabs.
By spraying on the skin with a chili cream (dermatologist) or aloe
one can usually make infestation areas visible, because then, as with
acne, small pustules etc, are created. Even with a black light lamp you
can see glassy fibers on the skin, which do not always come from
clothing abrasion. You can also apply milk or oils to the cleansed skin
to propel out some threads.
microscopic skin examinations by a light microscope and
skin scrapes, or an adhesive tape flap taken directly from the skin
surface, provide more clarity. Since usually some threads/fibers, as
well as a gel-like mass or granules stick to it. Typical infestation
sites can also be found in the areas of the eyes, nose, mouth and ears.
These samples should be cultivated, as mentioned above.
Also the eye pockets and cornea (cornea) and lens can be
the bacterial biofilm, by lymphatic discharge or by direct finger
contamination. Inflammation of the eyes (keratitis, uveitis) or
(conjunctivitis) may develop, with increased redness,
pain and a foreign body sensation in the eyes. Coupled with reduced
visual acuity and increased light shyness.
A survey of the patient about his or her circumstances would also be
useful in order to determine whether, for example, possible
via pets, partners, laundry, etc., should be ruled out.
All bacteria are located in a single biofilm, which in nature can
filamentously in the soil and twisted with fungal hyphae or plant roots
for a symbiosis. Hence the formation of filaments is related due to
stress because of a nitrogen deficiency or temperatures higher than 35
Unfortunately, such infections caused by various
agrobacteria (e.g. Rhizobium
radiobacter, B. megaterium, etc.) are still
unknown to most physicians and human laboratories, although there are
already sufficient medical case-studies.
Agrobacterium as human pathogen
Agrobacterium and relation to Morgellons
Common Plant Vector A. tumefaciens injects Genes into Human
Meanwhile, since first use in the 1980s, such agrobacteria
diagnosed more often as hospital germs worldwide, although according to
biotech companies that distribute them, their biological products are
not supposed to pose a danger to humans and animals. In other words,
collateral damage could occur in immunocompromised individuals.
This contradicts my findings, as most of the patients stated that they
did not actually have a noticeably weakened immune system. Rather they
traumatic and stressful times before the infection and some in turn
actually suffered from immunosuppressive pre-diseases such as cancer,
bacterial infections such as Lyme disease, etc. These diseases also
cause stress symptoms in the body with constant elevated stress
and histamine, but with lowered immunoglobulines.
See the following source information on agrobacterial hospital germs:
radiobacter, or otherwise called Agrobacterium tumefaciens
or A. radiobacter,
etc., so always the same bacterium only with other properties, can
produce systemic diseases, and is, besides Bacillus megaterium,
probably one of the main pathogens of this biofilm disease. B. megaterium can
cause antrax-like cutaneous wounds.
also Bacillus cereus,
which have been detected
more often in samples, initially cause blood poisoning or pertinent
inflammation, depending on the immune status. In the highest virulence
stage, it can even cause appendicitis. At a later stage with
insufficient treatment can also develop lung and joint inflammation,
bladder and sinus infections, etc., because the immune system
by filaments located there.
However, it is possible to check in advance whether there is also an
active infection by spirochetes. The fact is, if not all, e.g. Lyme
disease antigen bands are detectable, there can also be a completely
different bacteria that can cause cross reactions (band 41) by similar
antigens. But spirochetes are also used by the pesticide industry as
biological insecticides and the clinical symptoms could be interpreted
as Lyme disease!
BACTERIA, SPIROCHETES, AND RICKETTSIA AS INSECTICIDES
all sufferers sooner or later have lowered levels of vitamin B12,
vitamin D, serotonin, folic acid, iron, as well as a compromised
system (HPA stress axis) and demineralization, lack of iron and red
blood cells. Consequently, oxygen uptake is often disturbed and
anaemia and sleep disturbances (histamine excess) are also involved. As
the disease progresses, many suffer from chronic fatigue syndrome
tough and slimy cough, eye inflammation, blurred eyes, skin atrophies
and depigmentation areas, periodontitis.
Bridges, fillings and tooth loss, and often also muscle and joint
pain,oedema, nerve inflammation, low or elevated blood pressure,
forgetfulness, fibromyalgia, headache, eyelash and hair loss. Also with
the first signs of autoimmune diseases. This reinforces the conclusion
that such diseases can actually be caused by microbial infections.
is a lot of research on such diseases, but what happens when they are
also triggered by biofilms that cannot be easily detected?
In some sufferers, even after years, there are just few skin symptoms,
but only an infestation of the sinuses, nose, ears and especially the
eyes and surrounding tissue. A worse or blurred view is typical, with
later development towards cataracts.
2015: Recently, a British nurse was re-admitted after
successfully treating the Ebola virus, as the viruses had deposited
undetected in the vitreous swords of the eyes and then Ebola later
re-ignited. In parallel with many other viral or bacterial infections,
scientists have now discovered that Ebola is not only transmitted via
blood, etc., but also through body contact, e.g. skin sweat! This is
similar to Morgellons and many other infections.
In general, it can be said that cutaneous and subcutaneous
symptomsresult from biofilm filaments and integrated bacteria, which
lesions resulting from enzyme release. Many skin symptoms are not only
caused by Rhizobium
radiobacter, or Bazillus megaterium, B. cereus etc.,
but also overlap with secondary infections (opportunistic bacteria or
This biofilm can be divided into short filaments or small blebs and
washed through the lymphatic system into all physical areas and finally
into the skin pores, where there in the skin pores, develops into
filaments (coils, fuzzballs). Sometimes in the skin pores several
colourful filaments (tyrosinase-related colour production) are
and then appear as thread-balls, so-called black specks.
Since the biofilm adheres to the hair root, it grows out with the hair
on the skin surface in order to get back later into the skin by enzyme
release. This results in the typical allergic skin symptoms (pustules,
blisters). A short haircut is very helpful to reduce the adhered
Small biofilm filaments
and blebs growing out with hairs
Some filaments can also be transferred to other individuals or
objectsby sweating out on the skin through simple body contact.
is thus by sweat, skin and hair, blood, urine, tear fluid, stool, and
other bodily fluids!
Many visitors of my site claimed they likely have Morgellons.
However, they had no filaments that could be found anywhere, or typical
signs of biofilm infection, but similar skin symptoms (acne?).
skin symptoms or disease are related to Morgellons. A differential
diagnosis could be preceded by scabies and some other bacterial/mycotic
infectious skin diseases. These include in particular very similar
mycotic infections such as with Phaeoacremonium
Pityrosporum folliculitis or Coccidioides immitis and
As well as protozoa infections of e.B. Pythium insidiosum
diseases such as Prurigo or Urticaria, or endo- and subcutaneous
cygomycosis (mold infection), - these can cause similar skin symptoms.
histopathological examination is indicated in all cases.
Additional co-infections may occur later due to the multiplication of
opportunistic pathogens (candida, enterobacteria, viruses, etc.) due to
a partially weakened immune system (low immunoglobulins) also due to
kidney inflammation and cystitis. Immunoglobulins are increasingly
washed out by a constant urge to urinate. But also other pathogens may
be involved, which previously had contact with the mentioned
filaments/biofilm in nature, intestines, etc., and remained glued to
or were integrated into the biofilm.
traumatic experiences and dramatic strokes of fate are
characteristic for all sufferers. Mainly these earlier (post-traumatic
stress) and also current traumas, or latent infections and the
permanent stress hormone distributions along with physical consequences
(disbalancing of the vegetative nervous system and immune system,
pituitary insufficiency, HPA stress axis) are causative to get these
diseases with negative influence on the immune competence of the
diseases, multiple inflammations and, as a result, cancer
had developed in many patients in the late stages. 29% died of
aggressive pancreatic cancer!
But also a poor diet, or predispositional factors such as diabetes
mellitus, cancer and a previous, false corticosteroid therapy, nervous
diseases, viral diseases, pre-existing autoimmune diseases such as
Hashimoto, metabolic diseases, thyroid disease, as well as surgical
trauma, mercury fillings removals, can cause stress situations and be
decisive or trigger for infection with these pathogens. Increased
The biofilm can be first colonized in the mouth-nose area
as inthe scalp area or on the skin. A larger blister formation or a
occur only after 3-4 days or already on the first day of infestation,
depending on the sweat formation and the current virulence (food
deficiency of bacteria) of the biofilm filaments.
Often you can see skin lesions/pustules around the eye areas/head,
whereby also the eyebrows and eyelashes are infested and often also
fail.??? In the morning, the eyelashes are sometimes glued. Skin
tend to form more often at typical sweat zones.
Later also at other physical extremities, such as around the eyebrows,
eyelids and upper eyelids, eyelashes, ears, nose, lips, tongue, gums,
mucous membranes in the mouth and nose area, in the eyes, elbows,
nipples, knees, joints, feet, toes, vaginal area, cervical area,
scrotum. Also around the nipples, at the neck and at the
at the belt line, the shoulders, the back of the hand, between the
fingers and all other areas that produce sweat.
Some skin areas also resemble dermatoses or eczema, but the skin is
already massively infiltrated with the biofilm, which grows
predominantly subcutaneously and also constantly reacts to internal and
external influences, such as water, acids, alcohol, pressure. Also by
pressure points of the hands, which lie on the body or head or by
lying longer on a part of the body (side or shoulders, back), a sweat
layer is automatically formed in between, which also
influences the lymphatic system and there can manifest more skin
If these filaments are swallowed directly, the symptoms may be delayed
or even absent for a long time. Since these microorganisms then only
spread in the large and small intestine for the time being and later,
due to various factors, aim for an expansion towards the bloodstream or
subcutaneous areas and subsequently encapsulated in the nerve tissue or
connective tissue, mucous membranes, and much later after stress
conditions of the patient break out there again.
If we are talking about migrating biofilms, then possibly also resident
fungi (Candida) and enterobacteria, etc. could be dragged further into
the bloodstream, or involved in a DNA transfer to pass on resistance
According to an M-R-O survey, about 17% of sufferers (mostly
in warmer areas) are additionally burdened by infestations with various
insects, which also leads to initial misdiagnosis. Insects, as
is known, are increasingly attracted by nitrogen/ammonium.
When this bacterial biofilm settles in the human epidermis and the
glands, the metabolism of these nitrogen-binding bacteria (urease,
ammonium) may then attract more all sorts of insects and also leads
lay their eggs in the skin. At the same time, the nitrogen utilization
of these bacteria and the conversion (ammonium/ammonia) can lead to
nerve inflammation, cramps and nerve damage, etc.