WILL BE UPDATED/REVISED WITHIN JULY 2010

In the first initial phase after infection, diagnosis and recognition of these invasive fungal / bacterial organisms (Morgellons) is very difficult (See also "process of an infection"), since generally there is only a few typical skin symptoms recognizable and the overall clinical picture usually presents at first similar to "Scabies", or some fungal infections as "Phaeoacremonium parasiticum" or perhaps the protozoa "Pythium insidiosum".

The differential diagnosis should consider and exclude the usual pseudo-scabies, as well as any form of usual bacterial or other fungal skin infections. Infections with "Staphilococcus aureus" bacteria can show initially similar symptoms and they may be, sometimes, involved too. Worse are infections with MRSA (multi-resistant staphilococcus aureus) which leads mostly to death or amputations if not recognized and treated in the first days with newer or higher antibiotic treatments.

The immune competence of the patient also plays a role, of course, in the sort of physical expression of this infection. A weak immune system and a certain chemical body condition e.g. hormonal and granulozytes lack are playing a major role in immunity against parasites and invasive fungal infections. A decrease of total granulocyte counts in peripheral blood (below 1000 /mm3 ) has been recognized to be a crucial risk factor for the acquisition of similar fungal infections.

A former tick bite and infections with all typical involved bacterias (babesia, ehrlichea, rickettsia, anaplasma etc.), may be also a reason for a lower level of granulozytes, which is reducing in common the immunity against external parasitical infections, as well the morgellons infection.

But a direct substitution of granulocytes in immunocompromised patients has in most cases failed to prove efficacious. As well predisposing factors like diabetes mellitus, a former corticosteroid therapy, neutropenia, constant stress (cortisol), metabolism disorders, surgery trauma, and a malnutrition is mostly decisive for an occurrence of the pathogen`s symptomatology. But it can occur also in healthy patients in the presence of insignificant trauma.

Also the immune response to the antigens of this parasite occurs, basically, like that with other parasitic infection: induction phase, delayed hyper sensitivity, direct hyper sensitivity and sometimes later also with a desensitization.

The initial inoculation of parasites often takes place on hairy areas, or on bare skin patches. In both areas large abscess-like lesions, blister lesions, or pimple development, as well as skin rash can appear either within 3-14 days after exposure, or even during the first day of infestation. This is usually related to the relative size, the number and the different kinds of carried pathogens of the initial infesting parasites and if the contact happend over the skin or internally by swallowing.

It is possible, as well, that they can live undiscovered in the host body for a certain time. Some situation or a certain food can provoke them, like a trigger reaction such as with herpes, and then the manifestation of them is recognizable in typical skin symptoms. The difference between considerated "HEALTHY PEOPLE" and evidently infected persons might be, that in common some are reacting more allergical then others to such infections. Surely also healthy people may have morgellons but it takes longer to manifest skin or internal symptoms.

Previously one limited investigation on the predictability for infected areas between the fingers and the backs of the hand, may have led investigators to be circumspect in their physical exams for these types of infection.

This exam is surely not adequate for these new parasites, since the patient populations are NOT comparable since the study included mostly unsanitary, neglected individuals with a proven scabies infection.

Therefore, if there is suspicion for infection with this new parasite, the whole body should always be examined. This includes, but is not limited to: external upper arms, shoulder areas, lumbar region, inside thigh, as well as the nasal cavities, ear canals, etc. (see also process of infection)

Indeed many reports on this illness demonstrate initially a scabies-like behavior and skin distribution, with all typical preliminary signs, such as, even the skin scars that can be seen after an infestation.

If the pustules or nodular lesions frequently form around the eyebrows, upper and lower eyelids and on the forehead area, or in general in the facial area as well as on the scalp, especially in the nares and on the nose, and on occasion also throughout the whole body; then one can presume this to be an infestation of the new parasite, and therefore exclude any typical scabies infestation.

These first typical signs cannot count only as an indicator, but also as the propagation trend in the direction of scalp, face, ears and nose. Because in general also around the nipples, the backs of the hand, between fingers and all the other areas which generate sweat, will be the favorite areas where it can be found later.

The external symptoms particullary with an infestation on the hairs are noticeable that they will turning whitish and the hair will looks rather glassy and transparent as it turns grey in the usual form. It looks rather like an exhausted and transparent straw. It seems generally that this organisms prefers also the contents of the hairs (keratin, melanin).

Additionally a loss of sight (blur) and degradation of the eyes with inflammations/infections and increased luminous sensivity. A general poorer visibility is common due to the bacterial (keratitis) and mycotical biofilm (keratomycosis) on the retina (cornea). These eye symptoms infestation in the front and rear ocular segments, retina (temporary flashs/lightings) and on the cornea are caused from organisms which flow out of the lymph system.

However, there are connections to symptoms caused from fungi Endomorphthoraceae and Aspergillus spp. In general, all fungal symptoms can be compared to a mold infection.

Starting in general with lesions, pustules and nodules on the skin, as well with swelling of the lymph nodes, formation of wrinkles of the skin and pigment disturbances, cough, as well as numbness/tingling of the extremities and also an increasing visual disturbances of the eyes (cornea/callosity, double vision).

An acute illness runs first with chills or heat flushes. These symptoms are released also by the immune response of the human body. The outbreak of the chronic illness after an infection amounts on an average of 4-8 months or longer.

In the further process there occurs an encapsulation of the micro organisms which leads to connective tissue nodes with inflammatory granuloms, with edema, itch, papulosa exanthema and lymphadenitis. (see symptoms)

Folowed later with a chronic dermatitis, depigmentation of the skin (bright spots), lichenification and atrophy. Cellulitis and hanging skin and hanging lymph nodes can occur too, and a sclerosiatic lymphadenitisas.

Depigmentation pattern with doughy swellings, granulomas, inflammatory noduls/ indurations of skin tissue,

similar to a diffuse systemic scleroderma caused also from Borrelia burgdorferi infection.

In a later stage a general physical weakness is common, also a reduced efficiency, chronic fatigue syndrome accompanied by concentration problems and neurological signs as fibromyalgy, MS and meninigitis/encephalitis. As well a state of heightened anxiety, mood swings, angryness, resignation, depression, apathy and often suicide minds.

Watch also this video about lyme and co-infections

Most have also gastrointestinal difficulties (failure), unexplainable back pain, coughs, rigid neck and other parts and progressive parodontal disease (tooth decay) and gum detoriations.

Many cases include edemas, swollen legs, face etc. (lymph), later also swollen glands and lymph nodes and difficulty in breathing, heart and liver problems (insufficiency) and a constant weariness, as well opportunistic fungal infections (candida etc.) and urological problems.

Similar symptoms and particullary Meninigitis/Encephalitis might be caused not only from Lyme but also from viruses as Varizella, Coxsackie, Enterovirus, EBV (Ebstein-Barr Virus), Herpes simplex Typ 2, LCM-virus, HIV. As well from other bacteria such as Enderobacteria (E.coli), Mycoplasma, Clamydia pneu.,  Streptococcus group B, and Listeria monocytogenes. Also fungi (Aspergillus spp.) and parasites (Naegleria fowleri) can cause similar symptoms. 

(more information).

In a later stage of an infestation also an invasion (spiderweb-like slimy tubuli) of internal body areas can occur. (lung, nerves, organs, gut, brain). Different further complications can follow (thrombosis, heartache etc.).

More exact microscopic investigations result in clarity and are always compellingly necessary therefore. It is also always advisable if symptoms are lacking, if difficulty making the correct diagnosis, or recognition of the severity of the infestation, then one must receive a skin scrabing, or adhesive tape sampling, whatever results in better success in the recognition.

This should be especially helpful particullary if this specimen sample was taken directly at the infestation sites. Almost always there will be some external parasites and larvae, or ova (eggs) or at least unusual fibrous matter easly seen under microscop at 100-200X magnification.

However, one should know in addition what one actually should do with for which findings and what one must look for. If an scrabing or a withdrawal sample by needle brings a kind of coloured fuzz, then that maybe the only "parasite" you will be able to recover and you should accept this. Further samples could be attempted to try for more statistical significance, in order to exclude coincidences. Further measures should then be immediately initiated after positive findings.

Treatment Implementation

Especially on suspicion with these new parasites, usual and uncommon laboratory tests should be immediately initiated (Borrelia, Chlamydia pneu., Streptococcus pneu., Mycoplasma, Mycobacteria, Babesia, etc.). The lower table gives a small overview of the usual investigations and medications which were given normally by knowledgeable physicians. Particularly with Lyme, depending on infestation stages different antibiotic therapies are prescribed (see more under Lyme facts).

Don`t use Lindane, Perimethrin, Sulphur, or any other insecticidal cream, because the symptoms get worse.

Using the DNS standard test by means of the Polymerase-chain-reaction (PCR), until recently, PCR's have been used as a good means for detection of many un-identified parasitical infections, as well as their identification by means of certain customary blood tests (Sero-diagnostic). However, now some of these tests for the antigens of parasites have, unfortunately, become outdated because there are so many new strains and species of parasites and many parasites antigens were still not isolated yet.

One could also perform blood tests of these parasites with the use of recombinant antigens specifically directed against Protozoan, Helminthes, as well as other parasites of the internal organs. However, this could become also quite difficult, because presently there have been no antigens isolated from this multi life form. In addition, usual testing (Lyme) should occur at least 6 weeks after exposure, in order to insure adequate time for an immune response to be demonstrated on clinical test findings.

Even blood tests by Elisa test procedure, or using IgG, with Morgellons and its co-enzymes of infection (bacteria etc.) will not show always usual clinical findings, such as can happen with Borrelia (Lyme), the main pathogen bacteria of morgellons. Because scientists have already found out that the exciters can be encapsulated into human tissue or the exciters create their own cysts, to avoid detection and survive there inside the cysts for a prolonged time, and no clinical results can be found. For Lyme, better results have been found with the Bowen test or the Western Blot test technology used by "IGenex" of Palo Alto, CA.. To read more about this on the Internet, see www.igenex.com.

As well the very promising new intracellular LTT- Melisa test (Lymphozyt-Transformation-Test) is slowly becoming established. It bring more positive results then the other usual tests. It is measuring the cellular defence and shows always an exposition in intracellular stages and also if the infection is new or much older.

For being 95 % sure of a borrelia infection, the newest German test called T-Cellspot is actually the best on market. The T-Cellspot test with the Elispot-Assay (Enzyme Linked Immuno Spot Assay) is based on the research of antigene-specific zytokin-secretion through reactive Lymphozyts.

The T-Cellspot test is actually 200 time more sensitive than the older elisa test. As well it can show pretty soon after an infection (2-3 weeks) a seropositive result and therefore an earlier medical treatment can be started to prevent usual damage, due to a lack of quick recognition of borrelia. (read more here). For chronic lyme also the CD57 test can be used now.

At an advanced stage of infestation of the host organism after approximately 12-24 months, a reduction of protein/collagen and cellulitis might be present, but also an increased infestation of internal organs, the lymph system, the blood circulatory system, urinary and respiratory tract, or any other human body cavities and cells.

A rapid diagnosis and treatment in the initial phase of the infection, may prevent in any case the usual pathogensis. If allowed to progress a higher pathological damage is added.

Therefore an initial oral application (antifungal, antibiotica) or after an already longer existing infestation a subcutan antibiotic injection is more indicated, in order to work against a further disease process. Because the pathogenetic consequences of this invasion is unforeseen and most likely incurable and irrversible in further process (nerves, heart, liver and brain damage).

For an infected person each day counts in order to fight against the total invasion and plastification of the body. Antibiotics as Minocycline, Clarythromycin, Azithromycin, Roxytromycin or Bactrin are the first choice of recommanded treatments. In order to understand Morgellons, what we are fighting in the first instance, are any kind of entomopathogenic fungus and bacteria, such as borrelia, mycoplasma and opportunistic bacteria e.g. E. coli, Pseudomonas, Streptoccocus etc., and secondly opportunistic fungi (dermatophytes) and viruses, and surely the fiber remains. Perhaps various insectal infestations too, which been attracted from the Morgellons pheromones.

Antibiotics can`t be a permanent remedy, only in the beginning and should be reduced after a few month to prevent a damage of the liver and kidneys. Natural remedies should be added later too.

Since these life forms are pleo-vectors for multiple pathogens, some have been tested but not everybody have been proven with some of these viral, mycotical and bacterial or opportunistic involved pathogens, but most concerned have been infected with chlamydia, mycoplasma or "Borrelia burgdorferi" (Lyme).

Adeno-Virus

Actinomycetes

Giardia

Aspergillus

Acanthamoeba

WA-1 (Babesia-like)

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