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                  Tick bite                         Tick             Morgellons bites       Morgellon

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LYME DISEASE AND MORGELLONS (2007)
Almost all so called Morgellons symptoms are caused by a newly transmitted, or perhaps already present borreliosis (Lyme disease) and other bacteria. Sometimes with typical viral, protozoal and bacterial coinfections.

Which are caused from Anaplasma, Babesia (intracellular in red blood cells, anaemia, red pin prick dots on skin, dark urine), Bartonella (intracellular in granulozytes), Chlamydia, Rickettsia (meningo/encephalitis) Mycoplasma (respiratory, nerves, organs), and with the later rise of opportunistic infections (Candida, Epstein-Barr virus, cytomegalovirus, etc.).

Lyme disease which is caused by the spirochetal bacteria Borrelia burgdorferi s.s. (sensu lato), is not only transmitted by ticks, but also by other vectors, such as worms (nematodes), lice, bed bugs, mosquitoes, fleas, termites (symbiont: Borrelia recurrentis) and many other kinds of insect and arthropod vectors. These vectors can live in symbiosis with spirochetes and now, perhaps, with this unknown fungus-like organism (protozoan?) called Morgellons.

In any case, spirochetes are very extraordinary and higher developed bacteria. They are highly transformable (pleomorphic) and are found everywhere in nature. They particularly inhabit mud, dirt, soil and puddles, where they can settle in vectors mentioned above, living inside them in endosymbiosis or as ectosymbiont.

For example, many bacteria live inside the human gut, to digest our food. Termites and nematodes even farm spirochetes that together with protozoans (e.g. Mixotricha paradoxis) are involved to digest cellulose-based food for them, with their enzymes (e.g. cellulase).

The most harmless spirochetal variants are even found in the human mouth (oral spirochetes) and vaginal area (vaginal spirochetes), and other variants can be highly virulent, such as Treponema pallidum, which causes syphilis.

Check this video about highly organized plaque with spirochetes

Infection with the Borrelia genus of spirochete depends on whether and how these and other pathogens can enter directly into a wound. Usually, infection is a result of a bite from a bloodsucking vector, such as a tick, into which the contents of the vector's intestine can return flow into a host.

Another means of transmission is an insect or arthropod vector (e.g. mosquito) crushed onto a host's wound. The vector's saliva plays less of a role here, reducing the possibility of transmission, but virus or fungus contagion is possible.

Hidden Lyme disease:

Many people do not know that Lyme disease (Borrelia burgdorferi, spirochete bacteria) is contagious almost in the same manner as syphilis (Treponema pallidum, another spirochete bacteria) and can be also transmitted sexually.

Since Borrelia bacteria can invade all regions of the human body, it can be found not only in the bloodstream or as cystic forms (sleepers) in the connective tissue, but also in tears, sperm, urine, sputum and breast milk.

Open: Spirochete bacteria found in sperm

Open: Vaginal Spirochete infection

Open: More information about Borrelia spreading in the human body

Otherwise, how else could babies get Lyme disease if they were never bitten by any insect, and the same applies for some adults! Note: Lyme disease is not only transmitted by ticks, but also by nematodes, mosquitoes, spiders, mites, lice and fleas, and certainly by entomopathogenic fungi (used from pesticide industry) that eat ticks.

According to a recent study in Europe, 1-10% of mosquitoes carry Borrelia bacteria. It is just a Russian roulette in the summer as to whether one of ten mosquito bites can cause Lyme disease.

Statistics reveal that probably 35-40% or more of the population of many countries already carry Lyme bacteria in their bodies. Government and medical authorities try to hide these facts, because the cost for blood tests and treatments would bankrupt any health system.

Worldwide, governments would rather deny this disease almost completely, and a few countries will not even admit that Lyme disease is real. Many persons are not even aware that they already carry these and other parasites inside their bodies.

Infection and spreading:

After an infection a local skin reaction can happen and many times not. (See also Wikipedia and this study)

Later the Borrelia can spread further through the blood circulatory system and throughout the entire organism, striking each internal organ, the nervous system, all joints and tissues. This illness is also called multi-systemic.

At present exists three well-known types of this bacteria kind, “Borrelia sensu stricto” which is mainly present in the USA, “Borrelia afzelii” and “Borrelia garinii” are more present in Europe and Asia. It should be investigated by scientists if all Morggellons victims are suffering from the same subtype of borrelia!

All subtypes cause the same illness, but with different complications. B. garinii  e.g. leads to neurological complications and B.afzelii to skin complications, and so on. Generally all variants spreads throughout the lymph and circulatory system of the infestated organism.

Lyme runs quite typically in three phases. The individual`s  patient must not go through to all stages of Lyme, sometimes, quite spontaneous remission can happen, or there is between the first and second Lyme stage no time limit if the pathogens enters directly the bloodstream.

Particularly with Lyme depending upon stage or involved organs, different antibiotic therapies are advised. These antibiotic regiments should begin preventively upon suspicion of the disease, every day counts!

1. Stage

Beginning with a bite by a tick, the first stage of illness can last for a few days to several weeks. Surrounding the bite an inflammation appears which turns red on the outside with a clearer lighter center known as a bull-eye" rash. This inflammation continues to spread and may reach several inches daily and will be called “Erythema chronicum migrans”. It can also form a bluish, swollen skin areas (Lymphadenosis cutis benigna).

During the first 6 months of this infection stage, in booth cases, most suffer a general weakness and reducingness, headaches and pains of the joints and repeatedly from fever chills.

2. Stage

The second stage that arises after several weeks to months is characterized by neurological complaints. For example by inflammations of the brain skins (meningitis), the brain itself (encephalitis) or the nerve strands within the range of the spinal column (radiculitis).

Also face paralyses (facialisparesis) can occur and in the later process also the “Garin Bujadoux syndrome”. Which represents a painful inflammation of nerves and brain skins (meningopolyneuritis).

In most cases there arise asymmetrical paralyses, as well partly as sensitivity disturbances and swollen lymph nodes. Completely rarely there may also be inflammatory changes in the heart, with palpitations and common arise of the heart frequency.

3. Stage

In the third stage of Lyme which occurs after a few months or years, one can determine a slow fall of certain skin areas, which is called “Acrodermatitis chronica atrophicans. This occours on the scalp, arms, hands, legs and feet and perhaps the muscles can be involved in a later stage too.. The skin is very thinned out at the stretching sides.

In the later process in thrusts are formed chronic joint inflammations. This is called Lyme Arthritis and strikes predominantly the joints at the feet, elbows, knees and as well the fingers and toes. In the later disease process also chronic inflammations of the brain and the brain skin can be formed, which can cause a paralysis of the legs and arms.

Check this story what Lyme may cause to you! Lyme patient left alone by health care system!

http://rettetborrelioseopfermartin.npage.de/a_dramatic_lyme_story_37638549.html

 

Other Lyme information:

http://www.ilads.org/

http://www.lymedisease.org

http://www.lymedisease.org/news/lyme_disease_views/newlymetest.html

http://www.lymetap.com/lyme-disease-lab_tests

http://www.lymedisease.org/news/lyme_disease_views/newlymetest.html

http://www.thedestinlog.com/articles/medical-19223-clinic-patients.html

http://www.dovepress.com/articles.php?article_id=8224

http://lymedisease.org.au/news/sydney-morning-herald-article-on-lyme-disease-11-june-11-2/

http://www.bioresourceinc.com/downloads/lyme_conference032011.pdf

http://www.mentalhealthandillness.com/Articles/SexAndLymeDisease.htm

http://wn.com/Borrelia

Watch also this site from Dr Klinghard clinic and treatments

Open private German Lyme clinic site with all Lyme/Chlamydia/Mycoplasma tests

Open English Private Clinic which treats Lyme

Open Site of German lab in Hamburg CD-57 test for chronic Lyme

Open German T-Cellspot Lyme Test site

Short information on new T-Cell-Spot Test

Lyme diagnostics

Most Lyme tests show initially a negative result and then only a physical and neurological investigation (reflexes, muscle movement, sensitivity) a CT of the lymph nodes may show more, or if typical symptoms arise, such as rigidity of the neck, headache, joint pain, an so on.

To have better results one can try the new American Igenex test. Generally that test far exceeds the older test procedures. Also effective is sometimes LTT-Melisa (Lymphozyt-transformation-test).

Or the German test called T-Cellspot is actually one of the best on market. The T-Cellspot test with the Elispot-Assay is based on the research of antigene-specific cytokinsecretion through reactive lymphozyts. The T-Cellspot test is actually 200 time more sensitive than the older elisa test. Chronic lyme can be tested now with the CD-57 test.

Usual Lyme treatments

Generally one should begin in the initial stage with Doxycyclin during the first 2-3 months, alternatively with Amoxicillin and Minocyclin. If the joints are already involved, then one should extend the therapy. With simultaneous skin features in the initial stage, one can also take additionally Ampicillin or Macrolids, as Minocyclin, Chlarytromycin or Roxithromycin (throat/nasal/urinary/respiratory-system/clamydia) or Azytromycin.

Those that have experienced heart involvement or palpitations and those with an already longer existing illness, one may treat also with Cephalosporins (Penicillin based) . After 2-3 months one should accomplish an antigen measurement against certain components of the pathogens. During the therapy one should be able to determine a decrease of the antigens.

These newer medicines (macrolids, cephalosporins), are third generation antibiotics and can prevent also a disease outbreak. The chronic stage of Lyme (1-4 years) can be sometimes only treated with newer antibiotics as Rocephin or eventually Bicillin (syphilis antibiotic). Liver values should be monitored

At present no vaccines been developed. Since January 2006 the University of Wuerzburg (Germany) had success in vaccine development made of genetically modified tobacco plants (by transgene) which showed good results on mice. The former vaccine from N.Y (USA, 1999) was also very promising but it was removed suddenly from the market and this vaccine is used now only for animals.

Prognosis

The Lyme Lymphozytom and the Erythema migrans mostly heal with an antibiotic therapy, without consequences. With the advanced Acrodermatitis chronica atrophicans, a therapy does not always arrive at a complete restitution. The Arthritis heals approximately 80% of the cases after 3 months. Another 20% is to be expected after some years. 5% of the patients with a Facialisparese had residues.

With an acute Neuro-borreliosis the symptoms can be reduced with antibiotics. But with chronic Neuro-borreliosis, healing occours after months or may not occur at all. Even after a quite successful Lyme treatment some patient can develop a depressive syndrome or chronic fatigue syndrome or Fibromyalgia, which hardly responds then to a renewed antibiotic treatment.

This is not a typical subsequent depression after Lyme disease, but a typical reaction which can develop also with other heavy illnesses. After an appropriate differential diagnosis one can introduce also a therapy with certain tricyclic anti-depressives.

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M-R-O Author

DIAGNOSIS SYMPTOMS INFECTION PROCESS TREATMENTS LYME FACTS DELUSIONAL FACTS

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